By Heleen M. Oudemans-van Straaten, Lui G. Forni, A.B. Johan Groeneveld, Sean M. Bagshaw, Michael Joannidis
This functional consultant presents the reader with solutions to special clinically appropriate questions in regards to the overview and administration of acute kidney damage (AKI). All features of serious care nephrology are lined, from pathophysiology and analysis to prevention and remedy. The questions thought of relate to a variety of concerns, similar to: How do I diagnose AKI? How am i able to defend the kidney in scientific perform? How do I deal with sufferers with AKI? whilst may still I begin and the way do I practice renal substitute remedy (RTT)? which sort of RTT is ideal for my sufferer? may still I supply particular nutrition? as well as offering functional guidance and remedy algorithms, the ebook contains calculators for non-stop RRT and anticoagulant dosing. The authors are across the world popular specialists within the fields of extensive Care drugs and Nephrology and all contributions are written in a transparent and concise sort and feature been peer reviewed. Acute Nephrology for the serious Care Physician will function a truly important resource for intensivist internists, anesthesiologists and nephrologists serious about the administration and therapy of significantly sick sufferers prone to or suffering from AKI.
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Extra info for Acute Nephrology for the Critical Care Physician
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Acute kidney injury in the intensive care unit according to RIFLE. Crit Care Med. 2007;35(8):1837–43; quiz 52. 8. Cruz DN, Bolgan I, Perazella MA, et al. North East Italian Prospective Hospital Renal Outcome Survey on Acute Kidney Injury (NEiPHROS-AKI): targeting the problem with the RIFLE Criteria. Clin J Am Soc Nephrol. 2007;2(3):418–25. 9. Ostermann M, Chang R, Riyadh ICUPUG. Correlation between the AKI classification and outcome. Crit Care. 2008;12(6):R144. 10. Bagshaw SM, George C, Dinu I, Bellomo R.
The pathophysiology of chronic renal injury in these models may involve oxidative stress, alteration in gene expression, loss of peri-tubular capillaries , and the haemodynamic and fibrotic effects of angiotensin II . Thus, after AKI, initial chronic damage may be subtle and not be associated with obvious abnormality in serum biochemistry, but still of great prognostic significance. Importantly, management of risk factors may attenuate these pathophysiological processes and slow the progression of CKD.
Acute Nephrology for the Critical Care Physician by Heleen M. Oudemans-van Straaten, Lui G. Forni, A.B. Johan Groeneveld, Sean M. Bagshaw, Michael Joannidis